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I.  Peer Review Consultant for Medical Journals to Review Rosacea Articles

1.  Journal of Current Therapeutic Research
2. Clinical Therapeutics: The International Journal of Drug Therapy
3. Lasers in Surgery and Medicine

II.  Publications on Rosacea Treatment and Pathophysiology

1. Nase, G.P., and L. Sy. The red face of rosacea. Dr. Linda Sy Dermatologic Newsletter. 4(16): 1-4, October, 1999.
   
   
2.  Bitter, P., Sr. and Nase, G.P. Skin rejuvenation for sun damage, aging and rosacea using intense pulsed light. Lasers in Aesthetic Surgery. Keller, G., Lacombe, V., Lee, P., Watson, J.P., eds. Thieme Medical Publishers, NY, 2000.
   
3. Nase, G.P., Editorial Invite:  Rosacea Research Foundation:Sufferers Form Group to Improve Treatments for All.  Dermatology Times: The Leading Newsmagazine for Dermatologists. Issue: March 1, 2005, pgs. 1-3.
   
   
4. Nase, G.P., J. Jesitus: Latest Advances in Rosacea Treatment: Triple-Pass Laser Treatment.  Dermatology Times: The Leading Newsmagazine for Dermatologists. Issue: March 1, 2005, pgs. 11-14.
   
   
5. Nase, G.P., Editorial Invite:  Hope for Rosacea: Treatments for Facial Redness, Flushing and Triggers.  Dermatology Times: The Leading Newsmagazine for Dermatologists. Issue: (Submitted for Publication).
   
   
6. Nase, G.P., J. Darm: The Rosacea Research and Treatment Institute of Oregon: Research, Treatment & Collaboration.  Dermatology Times: The Leading Newsmagazine for Dermatologists. Issue: (In Process).

III.  Curriculum Vitae 

1. Education
2. Biomedical Research
3. Research Grants & Support
4. Awards & Honors
5. National & International Presentations
6. Research Seminars
7. Medical & Scientific Editorial Boards
8. Scientific Publications

IV.  What are my Qualifications:  What does a Medical Physiologist do?

1. What is a Medical Physiologist?
   

I.  Peer Review Consultant for Rosacea-Related Journals                       

1.  "Journal of Current Therapeutic Research"

•  Peer Reviewl Consultant (2001 - Current)
•  Review Clinical Research & Manuscripts on Rosacea
•  Review Manuscripts on Inflammatory Skin Disorders

m

http://www.currenttherapeuticres.com/js.html

 

 

2.  "Clinical Therapeutics:  Journal of Drug Therapy"

• Peer Review Consultant (2001 - Current)
• Review Clinical Research & Manuscripts on Rosacea
• Rosacea Article:  Volume 23, Number 10 
• Rosacea Article:  Volume 24, Number 1

m

http://www.clinicaltherapeutics.com/

3. "Lasers in Surgery and Medicine"

• Peer Review Consultant (2003 - Current)
• Review Medical Articles on the Effect of Lasers and Intense Pulse Light Systems on Rosacea Symptoms and Facial Flushing

m

Lasers in Surgery and Medicine

http://www3.interscience.wiley.com/cgi-bin/jhome/34073

II.  Curriculum Vitae

Education

1987-1991:

Pre-Medicine: B.S. in Biology, Concentration in Chemistry
Moravian College, Bethlehem, PA

1992-1997:

Ph.D. in Microvascular Physiology
West Virginia University School of Medicine
Department of Physiology, Morgantown, WV

1997-2002:

Post-Doctoral Fellowship
Indiana University School of Medicine
Department of Physiology, Indianapolis, IN

Biomedical Research Experience

1988-1992:

Clinical Laboratory Technician
Grandview Hospital, Clinical Laboratories, PA

1992-1997:

Ph.D. Candidate
West Virginia University School of Medicine, Morgantown, WV
Research Interests:

• Local & neural control of blood flow
• Sympathetic nerves and blood vessel receptors
• Endothelial cell and alpha-adrenergic receptor activation
• Technique-driven study -- Local endothelial cell damage via micropipette-injected CO2 embolization in vivo

1997-2002:

Post Doctoral Fellowship
Indiana University School of Medicine, Indianapolis, IN
Research Interests:

• Microvascular alterations in Type II Diabetes
• Microelectrode measurements of nitric oxide
• Endothelium-Derived Nitric Oxide Production during hypoxia and hyperglycemia

2002 - 2004:

Biomedical Research Scientist
Indiana University School of Medicine, Indianapolis, IN
Research Interests:

• Arteriolar Blood flow alterations in Type II Diabetes
• Role of Beta II PKC activation in Type II Diabetes and Insulin Resistance
• Microelectode measurements of arteriolar nitric oxide during blood flow perturbations

Research Grants and Support

1998-2000:

American Heart Association Grant #9804593

• "Nitric Oxide Regulation in the Microcirculation by Blood Flow and Oxygen Tension"
• Principal Investigator

1998-2004

National Institutes of Health Grant #HL-25824

• "Microvascular Characteristics of Diabetes Mellitus"
• Co-Investigator

Awards and Honors

• Dean's List for Academic Achievement, Moravian College, 1991
  
  
• Young Investigator Travel Award, Microcirculatory Society, Atlanta, GA, 1993
  
  
• National Dean's List for Academic Achievement, WVU School of Medicine, 1994-1997
  
  
• Van Liere Award for Excellence in Graduate/Medical Research, WVU School of Medicine, 1995
  
  
• Personal research selected for special discussion at International Vascular Biology Meeting, Germany, 1996
  
  
• Outstanding Young Investigator Travel Award, Microcirculatory Society, San Francisco, CA, 1998
  
  
• Outstanding Young Investigator Travel Award, Vascular Biology, Washington, DC, 1999
  
  
• Invited Speaker for National Vascular Biology Meeting, "Hypoxia Stimulates Endothelium-Derived Nitric Oxide", Orlando, FL, 2001

Societies and Memberships

• American Physiological Society, 1993-present
  
  
• Microcirculatory Society, 1996-present
  
  
• American Association for the Advancement of Science, 2000-present
  
  

Participation in National and International Meetings

• American Microcirculatory Society, Annual Meeting, Atlanta, GA, 1995
  
• American Microcirculatory Society, Annual Meeting, Bethesda, MD, 1996
  
• FASEB, Vascular Biology Annual Meeting, Washington, DC, 1996
  
• 6th World Congress Meeting for Microcirculation, Munich, Germany, 1996
  
• American Microcirculatory Society, Annual Meeting, New Orleans, LA, 1997
  
• 33rd International Congress of Physiological Sciences, St. Petersburg, Russia, 1997
  
• American Microcirculatory Society Annual Meeting, San Francisco, CA, 1998
  
• FASEB, Vascular Biology Annual Meeting, Washington, DC, 1999
  
• American Microcirculatory Society, Annual Meeting, Washington, DC, 1999
  
• FASEB, Vascular Biology Annual Meeting, San Diego, CA, 2000
  
• American Microcirculatory Society, Annual Meeting, San Diego, CA, 2000
  
• FASEB, Vascular Biology Annual Meeting, Orlando FL, 2001
  
• American Microcirculatory Society, Annual Meeting, FL, 2001
  
• American Microcirculatory Society, Annual Meeting, New Orleans, 2002
  
  
• American Microcirculatory Society, Annual Meeting, San Diego, CA, 2003

Research Seminars

• "Interaction Between Neural and Local Control Mechanisms in the Regulation of Microvascular Blood Flow", WVU School of Medicine, Dept. of Physiology, 1993
  
• "Nitric Oxide Modulates Arteriolar Responses to Increased Sympathetic Nerve Activity", WVU School of Medicine, Dept. of Physiology, 1994.
  
• "Blood Flow, Wall Shear Stress and Endothelium-Derived Relaxation", WVU School of Medicine, Dept. of Physiology, 1995.
  
• "The Microvascular Endothelium Attenuates Sympathetic Neurogenic Vasoconstriction in the Intestine", WVU School of Medicine, Van Liere Convocation Research Competition, 1995.
  
• "Control of Microvascular Blood Flow by an Endothelium - Derived Relaxing Factor", WVU School of Medicine, Dept. of Physiology, 1995.
  
• "Alpha-Adrenoceptors and the Microvascular Endothelium", Indiana University School of Medicine, Post-Doctoral Interview, 1996.
  
• "Endothelium-Derived Nitric Oxide Attenuates Sympathetic Neurogenic Constriction in the Intestinal Microcirculation", Dissertation Defense, WVU School of Medicine, 1997.
  
• "Acute Hyperglycemia Depresses Nitric Oxide Formation in the Spinotrapezius", Indiana University School of Medicine, Dept. of Physiology & Biophysics, 1997.
  
• "Microvascular Dysfunction in Type II Diabetes", Indiana University School of Medicine, Dept. of Physiology & Biophysics, 1998.
  
• "Hypoxia Stimulates Endothelium-Derived Nitric Oxide", Vascular Biology Meeting, FL, 2001.

Peer Review Consultant for Rosacea-Related Journal Articles

2001 - present:

• Peer Review Consultant for the Journal of Clinical Therapeutics (An International Peer-Reviewed Journal of Drug Therapy) to review and edit rosacea-related medical articles.

• Peer Review Consultant for Current Therapeutic Research to review and edit rosacea-related medical articles.
  

2003 - present:

• Peer Review Consultant for the medical journal, "Lasers in Surgery and Medicine" to review the effect of lasers and intense pulse light systems on rosacea symptoms and facial flushing.

Publications

Rosacea-Related Publications:

1. Nase, G.P., and L. Sy. The red face of rosacea. Linda Sy Dermatologic Newsletter. 4(16): 1-4, October, 1999.
   
   
2. Bitter, P., Sr. and Nase, G.P. Skin rejuvenation for sun damage, aging and rosacea using intense pulsed light. Lasers in Aesthetic Surgery. Keller, G., Lacombe, V., Lee, P., Watson, J.P., eds. Thieme Medical Publishers, NY, 2000.

3. Nase, G.P., Editorial Invite:  Rosacea Research Foundation:Sufferers Form Group to Improve Treatments for All.  Dermatology Times: The Leading Newsmagazine for Dermatologists. Issue: March 1, 2005, pgs. 1-3.
   
   
4. Nase, G.P., John Jesitus: Latest Advances in Rosacea Treatment: Triple-Pass Laser Treatment.  The Leading Newsmagazine for Dermatologists. Issue: March 1, 2005, pgs. 11-14.
   

5. Nase, G.P., Editorial Invite:  Hope for Rosacea: New Treatments on the Horizon for Rosacea Sufferers.  Dermatology Times: The Leading Newsmagazine for Dermatologists. Issue: (Submitted).
   

6. Nase, G.P., J. Darm: The Rosacea Research and Treatment Institute of Oregon: Research, Treatment & Collaboration.  Dermatology Times: The Leading Newsmagazine for Dermatologists. Issue: (Submitted).

Scientific Manuscripts:

1. Nase, G.P., and M.A. Boegehold. Nitric oxide modulates arteriolar responses to increased sympathetic nerve activity. Am. J. Physiol. 271 (Heart Circ. Physiol. 40): H860-H869, 1996.
   
   
2. Nase, G.P., and M.A. Boegehold. Modulation of sympathetic constriction by the arteriolar endothelium does not involve the cyclooxygenase pathway. Int. J. Microcirc. Clin. Exp. 17(1): 41-47, 1997.
   
   
3. Nase, G.P., and M.A. Boegehold. Endothelium-derived nitric oxide limits sympathetic neurogenic constriction in the intestinal microcirculation. Am. J. Physiol. 273 (Heart Circ. Physiol. 42): H426-H433, 1998.
   
   
4. Nase, G.P., and M.A. Boegehold. Postjunctional alpha 2-adrenoceptors are not present in proximal arterioles of rat intestine. Am. J. Physiol. 274 (Heart Circ. Physiol.): H202-H208, 1998.
   
   
5. J.M. Lash, G.P. Nase and H.G. Bohlen. Acute hyperglycemia depresses arteriolar nitric oxide formation in skeletal muscle. Am. J. Physiol. 277 (Heart Circ. Physiol.): H1513-H1520, 1999.
   
   
6. H.G. Bohlen, and G.P. Nase. Dependence of intestinal arteriolar regulation on flow mediated nitric oxide formation. Am. J. Physiol. 279 (Heart Circ. Physiol.): H2249-H2258, 2000.
   
   
7. H.G. Bohlen, and G.P. Nase. Arteriolar nitric oxide concentration is decreased during hyperglycemia-induced Beta-II PKC activation. Am. J. Physiol. (Heart Circ. Physiol.) 280(2): H621-H627, 2001.
   
   
8. H.G. Bohlen, and G.P. Nase. Obesity lowers hyperglycemic threshold for impaired in vivo endothelial nitric oxide function. Am. J. Physiol. (Heart Circ. Physiol.) 283(1): H391-H397, 2002.
   
   
9. H.G. Bohlen, G.P. Nase, and J.S. Jin. Invited Review: Multiple mechanisms of early hyperglycemic injury of the rat intestinal microcirculation. Clin. Exp. Pharmacol. Physiol 29(1-2):138-142, 2002.
   
   
10. Nase, G.P., J. Tuttle, and H.G. Bohlen. Reduced Perivascular PO2 increases nitric oxide release from endothelial cells. Am. J. Physiol (Heart Circ. Physiol.): 285: H507-H515, 2003.
    
    
11. H.G. Bohlen, and G.P. Nase. Insulin resistance predisposes arterioles to nitric oxide dysfunction during mild hyperglycemia. Am. J. Physiol. (Heart Circ. Physiol.): 2005 (Submitted).

Book Chapters:

1. Nase, G.P., and M.A. Boegehold. The influence of sympathetic nerves on microvascular tone is modulated by endothelial-derived nitric oxide. Proceedings of the 6th World Congress for Microcirculation, 1996.
   
   
2. Nase, G.P. Endothelium-derived nitric oxide attenuates sympathetic neurogenic constriction in the intestinal microvasculature. Dissertation, WVU School of Medicine, Dept. of Physiology, 1997.

Scientific Abstracts:

1. Nase, G.P., and M.A. Boegehold. Nitric oxide modulates arteriolar responses to increased sympathetic nerve activity. Microcirculation 2(1): 80, 1995.
   
   
2. Nase, G.P., and M.A. Boegehold. Endothelial-derived nitric oxide modulates sympathetic constriction in the intestinal microvasculature. Microcirculation 3(1): M127, 1996.
   
   
3. Nase, G.P., and M.A. Boegehold. Modulation of sympathetic constriction does not involve the cyclooxygenase pathway. FASEB J, 1996.
   
   
4. Nase, G.P., and M.A. Boegehold. The influence of sympathetic nerves on microvascular tone is modulated by endothelial-derived nitric oxide. Sixth World Congress for Microcirculation, Munich, Germany, 1996.
   
   
5. Nase, G.P., and M.A. Boegehold. Alpha adrenoceptors do not mediate nitric oxide release during increased sympathetic nerve activity. Microcirculation, 1997.
   
   
6. Boegehold, M.A., and G.P. Nase. Arteriolar sympathetic constriction is limited by endothelial nitric oxide via a mechanism that does not involve shear stress or alpha2-receptors. 33rd International Congress of Physiological Sciences, St. Petersburg, Russia, 1997.
   
   
7. Lash, J.M., G.P. Nase, and H.G. Bohlen. Acute hyperglycemia enhances dilation of rat spinotrapezius arterioles to sodium nitroprusside. Microcirculation, 1998.
   
   
8. Nase, G.P., J.M. Lash, and H.G. Bohlen. Periarteriolar nitric oxide concentration is depressed in rat skeletal muscle during hyperglycemia. FASEB J, 1999.
   
   
9. Bohlen, H.G., and G.P. Nase. In vivo intestinal arteriolar nitric oxide concentration is directly altered by blood flow velocity. Microcirculation, 1999.
   
   
10. H.G. Bohlen, and G.P. Nase. Beta-II protein kinase inhibition prevents and reverses in vivo endothelial nitric oxide suppression during acute hyperglycemia. Microcirculation, 2000.
    
    
11. Nase, G.P., and H.G. Bohlen. Arteriolar and venular nitric oxide concentration are increased during localized hypoxia. Microcirculation, 2000.
    
    
12. Nase, G.P., and H.G. Bohlen. Endothelium-derived nitric oxide production is enhanced during reduced oxygen availability. Microcirculation, 2001.
    
    
13. H.G. Bohlen, and G.P. Nase. Insulin resistance predisposes arterioles to nitric oxide dysfunction during mild hyperglycemia. Microcirculation, 2001.
    
    
14. H.G. Bohlen, and G.P. Nase. Arteriolar nitric oxide concentration is decreased during hyperglycemia-induced Beta-II PKC activation. Microcirculation, 2002.
    
    
15. H.G. Bohlen, and G.P. Nase. Obesity lowers hyperglycemic threshold for impaired in vivo endothelial nitric oxide function. Microcirculation, 2002.
    
    Nase, G.P., and H.G. Bohlen. Local hypoxia increases arteriolar and venular nitric oxide concentration in the intestinal microvasculature. Microcirculation, 2002.
    
    
16. H.G. Bohlen, and G.P. Nase. Insulin resistance predisposes arterioles to nitric oxide dysfunction during mild hyperglycemia. Microcirculation, 2002.

IV.  What is a Medical Physiologist?       

Brief Synopsis of Duties:

1. Perform scientific research to determine potential medications for human disease
   
   
2. Develop new medications to treat human disease
   
   
3. Perform scientific research on medication to determine cellular mechanisms, molecular mechansims, genetic interactions & safety profile
   
   
4. Administer drugs to patients in clinical trials & monitor effectiveness

Official Description of a Medical Physiologist with Post-Doctoral Training

Medical scientist jobs require a Ph.D. degree in a biological science. Biological scientists who do biomedical research are usually called medical scientists. Medical scientists work on basic research into normal biological systems to understand the causes of and to discover treatment for disease and other health problems. Medical scientists try to identify changes in a cell, chromosome, or even gene that signal the development of medical problems, such as different types of cancer. After identifying structures of or changes in organisms that provide clues to health problems, medical scientists work on the treatment of problems. For example, a medical scientist involved in cancer research may formulate a combination of drugs that will lessen the effects of the disease. Medical scientists with a medical degree can administer these drugs to patients in clinical trials, monitor their reactions, and observe the results. The medical scientist will return to the laboratory to examine the results and, if necessary, adjust the dosage levels to reduce negative side effects or to try to induce even better results. In addition to using basic research to develop treatments for health problems, medical scientists attempt to discover ways to prevent health problems from developing, such as affirming the link between smoking and increased risk of lung cancer, or between alcoholism and liver disease.

Biological and medical scientists usually work regular hours in offices or laboratories and usually are not exposed to unsafe or unhealthy conditions. Those who work with dangerous organisms or toxic substances in the laboratory must follow strict safety procedures to avoid contamination. Medical scientists also spend time working in clinics and hospitals administering drugs and treatments to patients in clinical trials.

In addition to formal education, medical scientists usually spend several years in a postdoctoral position before they apply for permanent jobs. Postdoctoral work provides valuable laboratory experience, including experience in specific processes and techniques, such as gene splicing, which are transferable to other research projects. In some institutions, the postdoctoral position can lead to a permanent position.

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